CEFALEA HORTON PDF

Esta saliendo al mercado una novedosa tecnologia especialmente hecha para aliviar los dolores de la Cefalea de Horton (Cefalea en Racimos-Cluster. Cluster headache (CH), also known as histamine headache, is a primary neurovascular headache disorder, the pathophysiology and etiology. Many translated example sentences containing “cefalea de Horton” – English- Spanish dictionary and search engine for English translations.

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Cluster headache CH is a neurological disorder characterized by recurrent severe headaches on one side of the head, typically around the eye. The cause is unknown. Recommended management includes lifestyle changes such as avoiding potential triggers. The condition cevalea about 0.

Cluster headache

Cluster headaches are recurring bouts of severe unilateral headache attacks. The onset of an attack is rapid and typically without cefaela aura. Preliminary sensations of pain in the general area of attack, referred to as “shadows”, may signal an imminent CH, or these symptoms may linger after an attack has passed, or between attacks.

The pain occurs only on one side of the head, around the eye, particularly above the eye, in the temple. The pain is typically greater than in other headache conditions, including migraines. The pain is typically described as burning, stabbing, drilling or squeezing, and may be located near or behind the eye. If left untreated, attack frequency may range from one attack every two days to eight attacks per day.

Restlessness for example, pacing or rocking back and forth may occur. Similar to a migraine, sensitivity to light photophobia or noise phonophobia may occur during a CH.

Nausea is a rare symptom although it has been reported. People with CH may dread facing another headache and adjust their physical or social activities around a possible future occurrence.

Likewise they may seek assistance to accomplish what would otherwise be normal tasks. They may hesitate to make plans because of the regularity, or conversely, the unpredictability of the pain schedule. These factors can lead to generalized anxiety disorders, panic disorder, [14] serious depressive hprton, [18] social withdrawal and isolation.

Cluster headaches may occasionally be referred to as “alarm clock headache” hprton of the regularity of their recurrence. CH attacks often awaken individuals from sleep.

Both individual attacks and the cluster grouping can have a metronomic regularity; attacks typically striking at a precise time of day each morning or night. The recurrence of headache cluster grouping may occur more often around solstices, or seasonal changes, sometimes showing circannual periodicity.

Conversely, attack frequency may be highly unpredictable, showing no periodicity at all. These observations have prompted researchers to speculate an involvement, or dysfunction of the hypothalamus. The Hypothalamus controls the body’s “biological clock” and circadian rhythm.

In accordance with the International Headache Society IHS diagnostic criteria, cluster headaches occurring in two or more cluster periods, lasting from 7 to days with a pain-free remission of one month or longer between the headache attacks, may be classified as episodic. If headache attacks occur for more than a year without pain-free remission of at least one month, the condition is classified as chronic.

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The frequency, severity and duration of headache attacks experienced by people during these cycles hortn between individuals and does not demonstrate complete remission of the episodic form. The condition may change unpredictably from chronic to episodic and from episodic to chronic. The cause of cluster headache is unknown. The vascular theory has been called into question [25] and other mechanisms are being considered.

Cluster headache may, but rarely, run in some families in an autosomal dominant inheritance pattern. A review suggests that the suprachiasmatic nucleus of the hypothalamuswhich is the major biological clock in the human body, may be involved in cluster headaches, because CH occurs with diurnal and seasonal rhythmicity. Positron emission tomography PET scans indicate the brain areas which are activated during attack only, compared to pain free periods. The area in the center in all three views is specifically activated during CH only.

The bottom row voxel-based morphometry VBM shows structural brain differences between individuals with and without CH; only a portion of the hypothalamus is different.

Cluster headaches are often misdiagnosed, mismanaged, or undiagnosed for many years; they may be confused with migraine, “cluster-like” headache hkrton mimicsCH subtypes, other TACs trigeminal autonomic cephalalgiasceralea other types of primary or secondary headache syndrome.

A detailed oral history aids practitioners in correct differential diagnosis, as there cefalra no confirmatory tests for CH. A headache diary can be useful in tracking when and where pain occurs, how severe it is, and how long the pain lasts. A record of coping strategies used may help distinguish between headache type; data on frequency, severity and duration of headache attacks are a necessary tool for initial and correct differential diagnosis in headache conditions.

Cefalea en racimos | Insight Medical Publishing

Correct diagnosis presents a challenge as the first CH attack may present where staff are not trained in the diagnosis of rare or complex chronic disease. Individuals with CH typically experience diagnostic delay before correct diagnosis. Cluster headache may be misdiagnosed as migraine or sinusitis. Incorrect terms like “cluster migraine” confuse headache types, confound differential diagnosis and are often the cause of unnecessary diagnostic delay, [40] ultimately delaying appropriate specialist treatment.

Preventive treatments are used to reduce or eliminate cluster headache attacks; they are generally used in combination with abortive and transitional techniques. The recommended first-line preventive therapy is verapamila calcium channel blocker. There is little evidence to support a long-term benefit from glucocorticoids[2] but they may be used until other medications take effect as they appear to be effective at three days.

Nerve stimulators may be an option in the small number of people who do not improve with medications. A number of cfalea procedures, such as a rhizotomy or microvascular decompressionmay also be considered, [47] but evidence to support cdfalea is limited and there are cases of people whose symptoms worsen after these procedures.

Cefalàlgia de Horton

Lithiummethysergideand topiramate are recommended alternative treatments, [45] [49] yorton there is little evidence supporting the use vefalea topiramate or methysergide. Treatment for cluster headache is divided into three primary categories: Oxygen therapy may help people with CH, but it does not help prevent future episodes.

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The other primarily recommended treatment of acute attacks is subcutaneous or intranasal cefzlea. The use of opioid cefaalea in management of CH is not recommended [55] and may make headache syndromes worse. The vasoconstrictor ergot compounds may be useful, [9] but have not been well studied in acute attacks.

Cluster headache affects about 0. The first complete description of cluster headache was given by the London neurologist Cfealea Harris inwho named the disease migrainous neuralgia. The cefale was originally named Horton’s cephalalgia after Bayard Taylor Hortona US neurologist who postulated the first theory as to their pathogenesis.

His original paper describes the severity of the headaches as being able to take normal men and force them to attempt or complete suicide; his paper said:. They had found no relief from the usual methods of treatment. Their pain was so severe that several of them horron to be constantly watched for fear of suicide. Most of them were willing to submit to any operation which might bring relief.

CH has alternately been called erythroprosopalgia of Bing, ciliary neuralgia, erythromelalgia of the head, Horton’s headache, histaminic cephalalgia, petrosal neuralgia, sphenopalatine neuralgia, vidian neuralgia, Sluder’s neuralgia, Sluder’s syndrome, and hemicrania angioparalyticia.

As of July [update] there are no approved medicines for the prevention of cluster headache in the United States.

Some controversial case reports suggest that ingesting tryptamines such as LSDpsilocybinor DMT can reduce pain and interrupt cluster headache cycles. Fremanezumaba humanized monoclonal antibody directed against calcitonin gene-related peptides fefalea and beta, is in phase 3 clinical trials for CH. From Wikipedia, the free encyclopedia. Cefwlea American Journal of Medicine.

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Case reports and a critical review of the literature”. Chronic paroxysmal hemicrania and Hemicrania continua. How they were discovered and what we have learned since”.